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Thursday, August 13, 2020 | History

3 edition of Transmural plasma flow in atherogenesis found in the catalog.

Transmural plasma flow in atherogenesis

Harry Jellinek

Transmural plasma flow in atherogenesis

by Harry Jellinek

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  • 1 Currently reading

Published by Akadémiai Kiadó in Budapest .
Written in

    Subjects:
  • Atherosclerosis -- Etiology.,
  • Blood-vessels -- Permeability.

  • Edition Notes

    StatementHarry Jellinek, Zoltán Detre, Béla Veress.
    ContributionsDetre, Zoltán., Veress, Béla.
    Classifications
    LC ClassificationsRC692 .J45 1983
    The Physical Object
    Pagination189 p. :
    Number of Pages189
    ID Numbers
    Open LibraryOL2917683M
    ISBN 109630532050
    LC Control Number84150177

      atherogenesis and focal distribution of atherosclerosis, suggesting that mechanical factors, The magnitude of the transmural flow is several orders of magnitude smaller than that of the bulk blood flow. Therefore, the time-scales characterising increasing filtration velocity, decreasing flow rate, and decreasing LDL plasma :// Drugs will be dispersed throughout the wall predominantly by transmural convection instead of diffusion if the Peclet number, Pe=J(1-ς f)/P, is greater than unity, where J is the transmural fluid flow per unit surface area and ς f and P are the reflection and permeability coefficients to the drug, respectively. Although the targets of local

    Platelets are anuclear cell fragments derived from the cytoplasm of BM megakaryocytes 1 that circulate in the bloodstream of humans for days. The primary hemostatic function of platelets has been recognized for more than a century, and the molecular processes regulating the adhesive function of platelets have been elucidated in great detail. 2–4 In addition to preventing excess bleeding Myocardial infarction or acute coronary syndromes, the actual term depending on the current definition 1 under which its various presentations are subsumed, remains the major clinical event in patients with atherosclerosis of the coronary arteries. 2. Besides its clinical presentation, the ECG is still the most important diagnostic tool in the emergency department. 3 While anterior and

      atherogenesis. Since the accumulation of plasma proteins such as low-density lipoprotein~LDL! in the artery wall is an important component of atherogenesis @4#, the effects of biomechanical forces on arterial wall permeability to these proteins is important. Specifically, the ratio of inner wall stress to outer wall [email protected]#, Case 3: The transmural flow across the RBC-free layer is modelled with a porous medium to simulate the glycocalyx, which is described by the Brinkman equation with a hydraulic permeability and a porosity of × 10 −18 and , respectively. The reaction of


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Transmural plasma flow in atherogenesis by Harry Jellinek Download PDF EPUB FB2

Full text Full text is available as a scanned copy of the original print version. Get a printable copy (PDF file) of the complete article (K), or click on a page image below to browse page by :// Sorry, our data provider has not provided any external links therefore we are unable to provide a link to the full :// Transmural Plasma Flow in Atherogenesis.

H.H.G. Jellinek. Published by Akademiai Kiado () ISBN ISBN Used. Hardcover. Good. This is an ex-library book and may have the usual library/used-book markings book has hardback covers. With owner's name and inscription inside cover. In good all round 1. Author(s): Jellinek,Harry; Detre,Zoltán; Veress,Béla Title(s): Transmural plasma flow in atherogenesis/ Harry Jellinek, Zoltán Detre, Béla :// Atherogenesis is the process of forming plaques in the intima layer of arteries.

Atherosclerosis is developed progressively with inflammation and lipid accumulation varying significantly among individuals. The accumulation of LDL and the inflammation of the arterial wall is the first stage of :// CiteSeerX - Document Details (Isaac Councill, Lee Giles, Pradeep Teregowda): Uptake of Low Density Lipoprotein (LDL) by the arterial wall is likely to play a key role in atherogenesis.

A particular process that may cause vascular scale heterogeneity in the rate of transendothelial LDL transport is the formation of a flow-dependent LDL concentration polarisation layer on the luminal surface of ?doi= Several lines of evidence supported the causative role of elevated plasma cholesterol in atherogenesis.

More recent studies have turned to studying the lipoproteins that transport cholesterol around the body, their interrelationships with diet and how they lead to :// Transmural pressures typically compress the SI at physiological pressures, which keeps this flow low.

Nguyen et al. () showed that aortic ECs express the water channel protein aquaporin-1 (AQP1) and the transEC (δP) portion of the transmural (ΔP) pressure difference drives, in parallel, water across AQP1s and plasma across interEC :// Transmural plasma flow in atherogenesis Jan 1, by Harry Jellinek Hardcover.

$ $ 9 Arterial Lesions and Arteriosclerosis Oct 1, by H. Jellinek Goodreads Book reviews & recommendations: IMDb Movies, TV & Celebrities: Weinbaum, S. Pfeffer, R. and Chien, S.,An hypothesis for the localization of atherogenesis and its relationship to fluid dynamics and transport.

in: “Proceedings of the International Symposium on the Role of Blood Flow in Atherogenesis”, Yoshida, Y. ed., Osaka. Google Scholar Jones CJH, Caro CG,The potential importance of arterial wall properties and blood flow in relation to atherogenesis in essential hypertension, Berlin, Biofluid Mechanics: Blood Flow in Large Vessels.

Proc. 2nd Int. Symp. Biofluid Mechanics and Biorheology, Publisher: Springer-Verlag, Pages: ?limit=30&id=&person=true&. The transport of macromolecules, such as low density lipoproteins (LDLs), across the artery wall and their accumulation in the wall is a key step in atherogenesis.

Our objective was to model fluid flow within both the lumen and wall of a constricted, axisymmetric tube simulating a stenosed artery, and to then use this flow pattern to study LDL Interestingly, such transient plasma membrane disruptions of aortic endothelial surface have been reported in vivo in those areas that have been subjected to a low mean shear stress and oscillatory flow with flow reversal.

The same areas are predilection places 45 Plasma apoE may gain access to the vessel wall in the context of small lipoprotein particles such as ␥-21 and pre-␤-LpE. 21 Plasma apoE is an important contributor to the ability of   Macromolecular transport across the vessel wall, apparently the earliest pre-atherosclerotic event, is known to occur due to advection by transmural (i.e., across the vessel wall) pressure-driven plasma (water and solutes) transport through leaky junctions around rare endothelial cells that are temporarily widened and leaky, some because they are either dying or :// This book contains 87 papers of the Symposium on mechanical aspects of blood flow and cardiovascular disease.

In the first section clinical studies deal with questions such as heart valve replacement surgical bypass techniques, ultrasound studies in humans and animals, the reaction of The Potential Importance of Arterial Wall Properties and Blood Flow in Relation to Atherogenesis in Essential Hypertension.

Pages Effect of the Transmural Pressure on LDL and Albumin Transport and Distribution Across the Intact Arterial Wall. Plasma Induced Surface Modification for Medical  › Physics › Classical Continuum Physics.

Flow Studies Group, Centre for Biological and Medical Sys­ tems, Imperial College of Science, Technology and Medicine, LondonSW72BY, UK. ©Edward Arnold The haemodynamic factors which have been most widely implicated in atherogenesis are those associated with blood flow, blood pressure and heart rate.

As will become evident later, these   A mean transmural pressure of mmHg, which in vivo is relatively high, was chosen to investigate the effect of hypertension on arterial LDL transport. Given that the transmural flow is several orders of magnitude smaller than the bulk flow, transmural pressure has a negligible effect on fluid dynamics in the blood :// L5, the most negatively charged subfraction of low-density lipoprotein (LDL), is implicated in atherogenesis, but the pathogenic association is relatively unexplored in patients with rheumatoid arthritis (RA).

We examined the role of L5 LDL in macrophage foam cell formation and the association of L5 with CD11c expression in THP-1 cells and RA patients. Using quantitative real-time PCR, we. To couple the flow dynamics in the arterial lumen with that in the arterial wall, the transmural velocity across the endothelium (J v) is modelled by the Kedem–Katchalsky equation where Δ p is the pressure difference across the endothelium, σ the osmotic reflection coefficient and Δ π the oncotic pressure drop across the ://  Title: A Cellular Scale Study of Low Denisty Lipoprotein Concentration Polarisation in Arteries: Authors: Vincent, P: Item Type: Thesis: Abstract:Plasma Induced Surface Modification for Medical Application.- Collapsible Tubes.- A Study of Predicted and Experimental Wall Collapse in Models of Highly Stenotic Arteries.- Pressure-Flow Relationships in Partially Occluded Flexible Tubes.- Flow in